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From age eight to eleven, I attended a primary school in Bath, England．It was a(n) 　　1　　 school composed of(由…组成) four classes with about 25 children in each class according to 　　2　　．For the most part, one teacher was responsible for teaching all subjects to the children in their class．　　3　　, sometimes the headmaster would come in and spend an hour or so, 　　4　　 some subject in which he was especially 　　5　　．The headmaster’s name was Mr．Ronald Broackes．　　6　　 he was quite strict about rules within the school, he had a sense of humor and would 　　7　　 telling the children small stories that would make us laugh．He was a very fair man and had a great influence on many of the children．In my own 　　8　　, I found that he took a great interest in me and he quickly discovered that I enjoyed 　　9　　．He would often stop me as I was going to class and produce a piece of paper from his pocket, often with a puzzle 　　10　　 on it．The puzzles were usually mathematical or logical(逻辑的)．As time went on, they slowly got more 　　11　　, but I loved them．Not only that, they kindled(点燃) within me a 　　12　　 of mathematics and problem-solving that stays with me to this 　　13　　．They also served to show me that intellectual activity was 　　14　　 when the correct answers were found, but perhaps more importantly it was great 　　15　　．To this day I can remember Mr．Broackes’ joyous cry of “Well done!” whenever I got a problem 　　16　　．This simple interaction with a man whom I 　　17　　 greatly has had a deep effect on my life．I shall forever be grateful for that．Mr．Broackes died just two weeks after the 　　18　　 that I had won the 1993 Nobel Prize in Physiology or Medicine．Unfortunately, I had no 　　19　　 to speak with him before he died．I learnt later that he had heard of my 　　20　　 and I will always hope that he realized the deep effect he had made on my life．

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阅读理解

　　“A key chemical of brain development may play a role in explaining why some people are genetically(由基因决定地)likely to suffer from anxiety and could lead to new treatments, ” the US researchers said．

　　They said highly anxious rats which were kept had very low levels of a brain chemical called growth factor FGF2, compared with the rats that were more relaxed．But when they improved the anxious rats’ living conditions－giving them new toys to play with and a bigger cage to live in-levels of this brain chemical increased and they became less anxious．

　　“The levels of this brain chemical increased in response to the experiences that the rats were exposed to．It also decreased their anxiety．” Javier Perez of the University of Michigan said in a telephone interview．“It made them behave the same way as the rats that were relaxed, ” he said．

　　In a former study of people who were severely depressed before they died, the team found the gene that makes FGF2 was producing very low levels of the growth factor, which is known primarily for organizing the brain during development and repairing it after injury．

　　Perez thinks the brain chemical may be a marker for genetic vulnerability(脆弱性)to anxiety and depression．But it can also respond to changes in the environment in a positive way, possibly by preserving new brain cells．

　　Although both the calm and anxious rats produced the same number of new brain cells, these cells were less likely to survive in the high-anxiety rats, the team found．Giving the rats better living conditions or injecting them with FGF2 helped improve cell survival．

　　“This discovery may help find the way for new, more specific treatments for anxiety that will not be based on sedation(药物镇静), but will instead fight the real cause of the disease, ” Dr．Pier Vincenzo Piazza from France said in a statement．

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